Infectious heart disease. Just like your average cold.

NIH-funded research sheds light on link between COVID-19 infection and increased risk of cardiovascular disease and stroke.

SARS-CoV-2, the virus that causes COVID-19, can directly infect the arteries of the heart and cause the fatty plaque inside arteries to become highly inflamed, increasing the risk of heart attack and stroke, according to a study funded by the National Institutes of Health. The findings(link is external), published in the journal Nature Cardiovascular Research, may help explain why certain people who get COVID-19 have a greater chance of developing cardiovascular disease, or if they already have it, develop more heart-related complications.

In the study, researchers focused on older people with fatty buildup, known as atherosclerotic plaque, who died from COVID-19. However, because the researchers found the virus infects and replicates in the arteries no matter the levels of plaque, the findings could have broader implications for anybody who gets COVID-19.

“Since the early days of the pandemic, we have known that people who had COVID-19 have an increased risk for cardiovascular disease or stroke up to one year after infection,” said Michelle Olive, Ph.D., acting associate director of the Basic and Early Translational Research Program at the National Heart, Lung, and Blood Institute (NHLBI), part of NIH. “We believe we have uncovered one of the reasons why.”

This study was done with the original strain.

Get vaccinated if you haven't yet.

Unvaccinated people who recover from Covid-19 at higher risk of heart complications: Study

  • FourteenEyes [he/him]
    ·
    edit-2
    1 year ago

    It's an endothelial disease and they should be emphasizing that shit like crazy

    The coughing is a side effect of the lining of your lungs' blood vessels inflaming

        • Spendrill@lemm.ee
          ·
          1 year ago

          Thanks for clarifying, also without your comment I wouldn't have learned a new and horrifying thing that it will be beneficial to know.

          • FourteenEyes [he/him]
            ·
            1 year ago

            See, you fucking corrected me and you hadn't heard its infection mechanism is primarily endothelial. It's fucking terrifying how effectively they've propagandized something that's still killing hundreds of Americans daily as not a big deal anymore and not worthy of further attention.

            • Spendrill@lemm.ee
              ·
              edit-2
              1 year ago

              I didn't correct you as much as ask for clarification, you obviously know more about this than I do. I never felt like it was just like getting a cold and when, after three vaccinations, I did get it despite it not being as hard on me as I had feared I never felt like it wasn't a big deal. There are definite long term effects, mainly cognitive, that are still happening. I did have a CAT scan to look for damage from smoking and I was fortunate enough to be told there was no sign of that and it happened after I had Covid so hopefully my respiratory and cardio-vascular systems have missed long term damage.

  • emizeko [they/them]
    ·
    1 year ago

    plaque inflammation doesn't worry me, because I don't have any teeth

  • autism_2 [any, it/its]
    ·
    1 year ago

    I don't mean this in a dismissive way, I'll still take proper precautions, but I've accepted the feeling that I'm probably not living past 60.

    • TheModerateTankie [any]
      hexagon
      ·
      1 year ago

      Vaccines have blunted a lot of the damage. Newer ones are in development that should help a lot more.

      But, yeah, people are getting infected multiple times a year with this thing. I'm betting on that being a bad thing that will shorten our lives. I don't think "building immunity" by repeated infection with a virus that can damage the immune system is logical course of action.